BPA and now the next three bisphenol family problems….
by Dr. Alan Kadish NMD
So now your well aware of the need to eliminate the BPA from your water bottle…..but you forgot to look at the other three bisphenol in this chemical family. All of the rest may prove to be worst than the original offender. The key take away from the two studies is their statement; ““This suggests there isn’t a safe bisphenol”.
If your noticing a bit of extra weight on your body consider that the hormonal effects of this chemical group also influence how we process our fat. It appears that they signal our fat cells to increase their storage, hence there name obesigens. Need more convincing ? How about the potential for cancers or changes in your kids development………enough said.
We are back to the original issue of ditching the plastics and moving forward with the many available alternatives. Consider light weight stainless steel or go for glass, with a silicon coat. Both offer the same convince of your other plastic water bottle but are non-toxic. When you look for a stainless bottle keep in mind that the spout should not be plastic, only silicon.
Now also keep in mind that the use of bisphenols is not limited to your water bottle….it’s in the lining of many canned food products to a host of consumer goods. So what’s a consumer to do…..start with decreasing your exposure by using lots less plastic products and make a habit of fresh food, forget the canned products. These simple steps will go a long way towards a more healthful life for you and your family.
Want the rest of the story ? We can do testing and evaluation of your current bodies burden of toxic chemicals and work to reduce your load and exposures. Call us at the Center of Health 541.773.3191
Effects of BPA Substitutes
Two studies add to the evidence that replacements for the plastic additive affect cells and animals in the same, untoward ways as bisphenol A.
April 11, 2016|
[paper] nicely shows is that not only BPA but a lot of BPA analogs really do have estrogenic activity,” said Deborah Kurrasch, a neuroscientist at the University of Calgary who has also examined the activity of BPS in zebrafish but was not involved in the study. “This suggests there isn’t a safe bisphenol.”
Last year, Kurrasch and colleagues reported that exposure to low doses of BPA or BPS cause larval zebrafish cells to mature into neurons prematurely. They also found that treated fish were more likely to display hyperactivity later on. “We did show there is some consequence to this precocious neurogenesis,” she said.
In December 2015, Nancy Wayne, who studies reproduction at the University of California, Los Angeles, and her collaborators reported the effects of BPS and BPA on the zebrafish reproductive system. And the team found similar results: exposure to both chemicals resulted in greater numbers of reproductive neurons and an upregulation of reproduction-related genes, the researchers reported in Endocrinology. “If you look at the structures of BPA and BPS, they’re so similar that it would be surprising if there weren’t similar effects for everything you look at,” Wayne said.
And the effects don’t stop at the neuroendocrine system.
Another study published last month (March 22) in Endocrinology looked instead at the influence of BPS on fat production. Exposing cell precursors extracted from women to BPA and BPS, Ella Atlas of Health Canada and colleagues found that the chemicals led the cells to accumulate lipids and increase the level of transcripts indicative of differentiation into fat cells. “They both seem to increase adipogenesis,” Atlas told The Scientist, with BPS being more potent than BPA.
Atlas’s experiments suggested that BPS acts through a fatty acid receptor called PPARγ, which controls fat cell development. A number of environmental chemicals suspected of being “obesogens,” meaning they may cause weight gain, also activate PPARγ.
“We have a problem of obesity in this country and it’s been blamed entirely on a change in our level of physical activity and our diet, and it certainly is believable,” said Wayne. “But there can be in addition to that another culprit. And another culprit can be various endocrine-disrupting chemicals that are altering metabolism.”
Atlas said it’s impossible to extrapolate from her study how BPS affects people, and whether it causes fat production in humans in vivo. “It’s basically raising a flag that it may be a problem,” she said.
Anne Marie Vinggaard of the National Food Institute at the Technical University of Denmark has also looked at lipid accumulation in fat cells, finding that BPS and BPA act similarly. “So far we think the data are indicating that we should not substitute BPA with BPS,” she said.
Yet BPA continues to be the prime focus of research and policy efforts to protect consumers while its analogs fly under the radar. Last month, the European Union announced that it is considering new restrictions on BPA in food packaging—specifically, how much of the chemical would be allowed to migrate from containers to food. But there is no mention of BPS, BPF, or any other similar substance that may be present.
“Most people think, ‘BPA-free, oh, it’s safe,’” said Wayne. “What does that mean? If they’re not using BPA, what are they using? And is it safer?”
J. Cano-Nicolau et al., “Estrogenic effects of several BPA analogs in the developing zebrafish brain,” Frontiers in Neuroscience, doi:10.3389/fnins.2016.00112, 2016.
J.G. Boucher et al., “Bisphenol S induces adipogenesis in primary human preadipocytes from female donors,” Endocrinology, 157:1397–1407, 2016.
W. Qiu et al., “Actions of bisphenol A and bisphenol S on the reproductive neuroendocrine system during early development in zebra
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